Fight-or-flight response explained

The fight-or-flight or the fight-flight-freeze-or-fawn[1] (also called hyperarousal or the acute stress response) is a physiological reaction that occurs in response to a perceived harmful event, attack, or threat to survival.[2] It was first described by Walter Bradford Cannon in 1915.[3] His theory states that animals react to threats with a general discharge of the sympathetic nervous system, preparing the animal for fighting or fleeing.[4] More specifically, the adrenal medulla produces a hormonal cascade that results in the secretion of catecholamines, especially norepinephrine and epinephrine.[5] The hormones estrogen, testosterone, and cortisol, as well as the neurotransmitters dopamine and serotonin, also affect how organisms react to stress.[6] The hormone osteocalcin might also play a part.[7] [8]

This response is recognised as the first stage of the general adaptation syndrome that regulates stress responses among vertebrates and other organisms.[9]

Name

Originally understood as the "fight-or-flight" response in Cannon's research,[3] the state of hyperarousal results in several responses beyond fighting or fleeing. This has led people to calling it the "fight, flight, freeze" response, "fight-flight-freeze-fawn" or "fight-flight-faint-or-freeze", among other variants.[10] The wider array of responses, such as freezing, fainting, fleeing, or experiencing fright,[11] has led researchers to use more neutral or accommodating terminology such as "hyperarousal" or the "acute stress response".

Physiology

Autonomic nervous system

See also: Autonomic nervous system. The autonomic nervous system is a control system that acts largely unconsciously and regulates heart rate, digestion, respiratory rate, pupillary response, urination, and sexual arousal. This system is the primary mechanism in control of the fight-or-flight response and its role is mediated by two different components: the sympathetic nervous system and the parasympathetic nervous system.[12]

Sympathetic nervous system

See also: Sympathetic nervous system. The sympathetic nervous system originates in the spinal cord and its main function is to activate the arousal responses that occur during the fight-or-flight response.[13] The sympathetic nervous system transfers signals from the dorsal hypothalamus, which activates the heart, increases vascular resistance, and increases blood flow, especially to the muscle, heart, and brain tissues.[14] It activates the adrenal medulla, releasing catecholamines that amplify the sympathetic response. Additionally, this component of the autonomic nervous system utilizes and activates the release of norepinephrine by the adrenal glands in the reaction.[15]

Parasympathetic nervous system

The parasympathetic nervous system originates in the sacral spinal cord and medulla, physically surrounding the sympathetic origin, and works in concert with the sympathetic nervous system. It is known as the calming portion of the autonomic nervous system.[16] While the sympathetic nervous system is activated, the parasympathetic nervous system decreases its response. Efferent vagal fibers originating from the nucleus ambiguous fire in parallel to the respiratory system, decreasing the vagal cardiac parasympathetic tone.[17] After the fight or flight response, the parasympathetic system's main function is to activate the "rest and digest" response and return the body to homeostasis. This system utilizes and activates the release of the neurotransmitter acetylcholine.[18]

Reaction

The reaction begins in the amygdala, which triggers a neural response in the hypothalamus. The initial reaction is followed by activation of the pituitary gland and secretion of the hormone ACTH.[19] The adrenal gland is activated almost simultaneously, via the sympathetic nervous system, and releases the hormone epinephrine. The release of chemical messengers results in the production of the hormone cortisol, which increases blood pressure, blood sugar, and suppresses the immune system.[20]

The initial response and subsequent reactions are triggered in an effort to create a boost of energy. This boost of energy is activated by epinephrine binding to liver cells and the subsequent production of glucose.[21] Additionally, the circulation of cortisol functions to turn fatty acids into available energy, which prepares muscles throughout the body for response.[22]

Catecholamine hormones, such as adrenaline (epinephrine) or noradrenaline (norepinephrine), facilitate immediate physical reactions associated with a preparation for violent muscular action.[23]

Function of physiological changes

The physiological changes that occur during the fight or flight response are activated in order to give the body increased strength and speed in anticipation of fighting or running. Some of the specific physiological changes and their functions include:[24] [25] [26]

Emotional components

Emotion regulation

See also: Emotional self-regulation. In the context of the fight or flight response, emotional regulation is used proactively to avoid threats of stress or to control the level of emotional arousal.[27] [28]

Emotional reactivity

During the reaction, the intensity of emotion that is brought on by the stimulus will also determine the nature and intensity of the behavioral response.[29] Individuals with higher levels of emotional reactivity (Such as an anxiety disorder) may be prone to anxiety and aggression, which illustrates the implications of appropriate emotional reaction in the fight or flight response.[30] [31]

Cognitive components

Content specificity

The specific components of cognitions in the fight or flight response seem to be largely negative. These negative cognitions may be characterised by: attention to negative stimuli, the perception of ambiguous situations as negative, and the recurrence of recalling negative words.[32] There also may be specific negative thoughts associated with emotions commonly seen in the reaction.[33]

Perception of control

See also: Control (psychology). Perceived control relates to an individual's thoughts about control over situations and events.[34] Perceived control should be differentiated from actual control because an individual's beliefs about their abilities may not reflect their actual abilities. Therefore, overestimation or underestimation of perceived control can lead to anxiety and aggression.[35]

Social information processing

See also: Social information processing (cognition). The social information processing model proposes a variety of factors that determine behavior in the context of social situations and preexisting thoughts.[36] The attribution of hostility, especially in ambiguous situations, seems to be one of the most important cognitive factors associated with the fight or flight response because of its implications towards aggression.[37]

Other animals

Evolutionary perspective

An evolutionary psychology explanation is that early animals had to react to threatening stimuli quickly and did not have time to psychologically and physically prepare themselves.[38] The fight or flight response provided them with the mechanisms to rapidly respond to threats against survival.[39] [40]

Examples

A typical example of the stress response is a grazing zebra. If the zebra sees a lion closing in for the kill, the stress response is activated as a means to escape its predator. The escape requires intense muscular effort, supported by all of the body's systems. The sympathetic nervous system's activation provides for these needs. A similar example involving fight is of a cat about to be attacked by a dog. The cat shows accelerated heartbeat, piloerection (hair standing on end), and pupil dilation, all signs of sympathetic arousal.[23] Note that the zebra and cat still maintain homeostasis in all states.

In July 1992, Behavioral Ecology published experimental research conducted by biologist Lee A. Dugatkin where guppies were sorted into "bold", "ordinary", and "timid" groups based upon their reactions when confronted by a smallmouth bass (i.e. inspecting the predator, hiding, or swimming away) after which the guppies were left in a tank with the bass. After 60 hours, 40 percent of the timid guppies and 15 percent of the ordinary guppies survived while none of the bold guppies did.[41] [42]

Varieties of responses

Animals respond to threats in many complex ways. Rats, for instance, try to escape when threatened but will fight when cornered. Some animals stand perfectly still so that predators will not see them. Many animals freeze or play dead when touched in the hope that the predator will lose interest.

Other animals have alternative self-protection methods. Some species of cold-blooded animals change color swiftly to camouflage themselves.[43] These responses are triggered by the sympathetic nervous system, but, in order to fit the model of fight or flight, the idea of flight must be broadened to include escaping capture either in a physical or sensory way. Thus, flight can be disappearing to another location or just disappearing in place, and fight and flight are often combined in a given situation.[44]

The fight or flight actions also have polarity – the individual can either fight against or flee from something that is threatening, such as a hungry lion, or fight for or fly towards something that is needed, such as the safety of the shore from a raging river.

A threat from another animal does not always result in immediate fight or flight. There may be a period of heightened awareness, during which each animal interprets behavioral signals from the other. Signs such as paling, piloerection, immobility, sounds, and body language communicate the status and intentions of each animal. There may be a sort of negotiation, after which fight or flight may ensue, but which might also result in playing, mating, or nothing at all. An example of this is kittens playing: each kitten shows the signs of sympathetic arousal, but they never inflict real damage.

Further reading

Notes and References

  1. Book: Walker, Peter . 2013 . Complex PTSD: From Surviving to Thriving : a Guide and Map for Recovering from Childhood Trauma . An Azure Coyote Book. 978-1-4928-7184-2.
  2. Book: Cannon, Walter. Wisdom of the Body. 1932. W.W. Norton & Company. United States. 978-0-393-00205-8.
  3. Book: Bodily changes in pain, hunger, fear, and rage. Walter Bradford Cannon. Appleton-Century-Crofts. 1915. New York. 211.
  4. Jansen. A. Nguyen, X . Karpitsky, V . Mettenleiter, M . Central Command Neurons of the Sympathetic Nervous System: Basis of the Fight-or-Flight Response. Science Magazine. 27 October 1995. 5236. 270. 644–6. 10.1126/science.270.5236.644. 7570024. 1995Sci...270..644J. 38807605.
  5. Book: Walter Bradford Cannon . Bodily Changes in Pain, Hunger, Fear and Rage: An Account of Recent Researches into the Function of Emotional Excitement . 1915 . .
  6. News: Adrenaline, Cortisol, Norepinephrine: The Three Major Stress Hormones, Explained. 16 August 2014. Huffington Post. April 19, 2014.
  7. Web site: Kwon. Diana. Fight or Flight May Be in Our Bones. 2020-06-22. Scientific American. en.
  8. Web site: Bone, not adrenaline, drives fight or flight response. 2020-06-22. phys.org. en.
  9. Book: Gozhenko, A. PATHOLOGY – Theory. Medical Student's Library. 2009. Radom. 270–275. Gurkalova, I.P. . Zukow, W . Kwasnik, Z .
  10. Book: Donahue . J.J. . Fight-Flight-Freeze System . Zeigler-Hill . V. . Shackelford . T.K. . Encyclopedia of Personality and Individual Differences . 2020 . 1590–1595 . 10.1007/978-3-319-24612-3_751 . 978-3-319-24610-9 . 240856695 .
  11. Bracha . H. Stefan . Freeze, Flight, Fight, Fright, Faint: Adaptationist Perspectives on the Acute Stress Response Spectrum . CNS Spectrums . September 2004 . 9 . 9 . 679–685 . 10.1017/S1092852900001954 . 15337864 . 8430710 . 31 May 2021.
  12. Book: Janig. W. Human Physiology. 1989. Springer-Verlag. New York, NY. 333–370. Schmidt, A . Thews, G . 2. Autonomic Nervous System.
  13. Book: Myers . David G. . Psychology . DeWall . C. Nathan . MacMillan Publishing . 2021 . 13 . 422.
  14. Kozlowska . Kasia . Walker . Peter . McLean . Loyola . Carrive . Pascal . 2015 . Fear and the Defense Cascade: Clinical Implications and Management . . 23 . 4 . 263–287 . 10.1097/HRP.0000000000000065 . 1067-3229 . 4495877 . 26062169.
  15. Web site: Chudler . Eric . Neuroscience For Kids . 19 April 2013 . University of Washington.
  16. Book: Myers . David G. . Psychology . DeWall . C. Nathan . MacMillan Publishing . 2021 . 13 . 422.
  17. Kozlowska . Kasia . Walker . Peter . McLean . Loyola . Carrive . Pascal . 2015 . Fear and the Defense Cascade: Clinical Implications and Management . . 23 . 4 . 263–287 . 10.1097/HRP.0000000000000065 . 1067-3229 . 4495877 . 26062169.
  18. Web site: Chudler . Eric . Neuroscience For Kids . 19 April 2013 . University of Washington.
  19. Web site: Margioris . Andrew . ACTH Action on the Adrenal . Endotext.org . 18 April 2013 . Tsatsanis, Christos . April 2011 . dead . https://web.archive.org/web/20130306182338/http://www.endotext.org/adrenal/adrenal5/adrenal5.htm . 6 March 2013 .
  20. Padgett. David. Glaser, R. How stress influences the immune response. Trends in Immunology. August 2003. 24. 8. 444–448. 10.1016/S1471-4906(03)00173-X. 12909458. 10.1.1.467.1386.
  21. Web site: King . Michael . PATHWAYS: GLYCOGEN & GLUCOSE . Washington University in St. Louis.
  22. Web site: HOW CELLS COMMUNICATE DURING THE FIGHT OR FLIGHT RESPONSE . University of Utah . 18 April 2013 . dead . https://web.archive.org/web/20130808004906/http://learn.genetics.utah.edu/content/begin/cells/fight_flight/ . 8 August 2013 .
  23. Book: . Psychology . 6 . 2004 . . 978-0-393-97767-7 .
  24. Web site: Stress Management for Health Course . The Fight Flight Response . 19 April 2013.
  25. Web site: Olpin . Michael . The Science of Stress . dead . https://web.archive.org/web/20171120215838/http://faculty.weber.edu/molpin/healthclasses/1110/bookchapters/stressphysiologychapter.htm . 2017-11-20 . 2013-04-25 . Weber State University.
  26. Book: Myers . David G. . Psychology . DeWall . C. Nathan . MacMillan Publishing . 2021 . 13 . 422.
  27. Cistler. Josh. Bunmi O. Olatunji . Matthew T. Feldner . John P. Forsyth . Emotion Regulation and the Anxiety Disorders: An Integrative Review. Journal of Psychopathology and Behavioral Assessment. 2010. 32. 1. 68–82. 10.1007/s10862-009-9161-1. 20622981. 2901125.
  28. Gross. James. Sharpening the Focus: Emotion Regulation, Arousal, and Social Competence. Psychological Inquiry. 1998. 9. 4. 287–290. 10.1207/s15327965pli0904_8.
  29. Avero. Pedro. Calvo, M. Emotional reactivity to social-evaluative stress: genderdifferences in response systems concordance. Personality and Individual Differences. 1 July 1999. 27. 1. 155–170. 10.1016/S0191-8869(98)00229-3.
  30. Carthy. T. Horesh N . Apter A . Edge MD . Gross JJ . Emotional reactivity and cognitive regulation in anxious children. Behaviour Research and Therapy. May 2010. 48. 5. 384–393. 10.1016/j.brat.2009.12.013. 20089246. 14382059.
  31. Valiente. C. Eisenberg N . Smith CL . Reiser M . Fabes RA . Losoya S . Guthrie IK . Murphy BC . The relations of effortful control and reactive control to children's externalising problems: A longitudinal assessment. Personality. December 2003. 71. 6. 1171–1196. 10.1111/1467-6494.7106011 . 14633062.
  32. Reid. Sophie C.. Salmon. Karen. Peter F. Lovibond. Cognitive Biases in Childhood Anxiety, Depression, and Aggression: Are They Pervasive or Specific?. Cognitive Therapy and Research. October 2006. 30. 5. 531–549. 10.1007/s10608-006-9077-y. 28911747.
  33. Book: Beck, Aaron. Cognitive Therapy and the Emotional Disorders. 1979. Penguin Books. United States.
  34. Weems. CF. Silverman, WK. An integrative model of control: implications for understanding emotion regulation and dysregulation in childhood anxiety. Journal of Affective Disorders. April 2006. 91. 2. 113–124. 10.1016/j.jad.2006.01.009. 16487599.
  35. Brendgen. M. Vitaro F . Turgeon L . Poulin F . Wanner B . Is there a dark side of positive illusions? Overestimation of social competence and subsequent adjustment in aggressive and nonaggressive children. Journal of Abnormal Child Psychology. June 2004. 32. 3. 305–320. 10.1023/B:JACP.0000026144.08470.cd. 15228179. 11239252.
  36. Crick. Nicki. Dodge, Kenneth. A review and reformulation of social information-processing mechanisms in children's social adjustment. Psychological Bulletin. January 1994. 115. 1. 74–101. 10.1037/0033-2909.115.1.74.
  37. Dodge. Kenneth. Social cognition and children's aggressive behavior. Journal of Child Development. March 1980. 51. 1. 162–170. 10.2307/1129603. 1129603. 7363732 .
  38. Adamo . S. A. . 2014-09-01 . The Effects of Stress Hormones on Immune Function May be Vital for the Adaptive Reconfiguration of the Immune System During Fight-or-Flight Behavior . Integrative and Comparative Biology . en . 54 . 3 . 419–426 . 10.1093/icb/icu005 . 1540-7063. free . 24691569 .
  39. Web site: Grohol. John. What's the purpose of the fight or flight response?. 18 April 2013. 23 March 2013. https://web.archive.org/web/20130323170934/http://psychcentral.com/blog/archives/2012/12/04/whats-the-purpose-of-the-fight-or-flight-response/. dead.
  40. Goldstein. David. Kopin, I. Evolution of concepts of stress. Stress. 2007. 10. 2. 10.1080/10253890701288935. 109–20. 17514579. 25072963. free.
  41. Dugatkin. Lee Alan. Tendency to inspect predators predicts mortality risk in the guppy (Poecilia reticulata). Behavioral Ecology. 1992. Oxford University Press. 3. 2. 125–127. 10.1093/beheco/3.2.124. September 9, 2020.
  42. Book: Nesse. Randolph. Randolph M. Nesse. Williams. George C.. George Christopher Williams. Why We Get Sick: The New Science of Darwinian Medicine. 1994. 213. Vintage Books. New York. 978-0-679-74674-4.
  43. Book: Gill, A.C.. Revision of the Indo-Pacific dottyback fish subfamily Pseudochrominae (Perciformes: Pseudochromidae). 2004. Smithiana Monographs. 1–123.
  44. Singh. J. Aballay, A. Microbial Colonization Activates an Immune Fight-and-Flight Response via Neuroendocrine Signaling. Developmental Cell. April 8, 2019. 49. 1. 89–99. 10.1016/j.devcel.2019.02.001. 30827896. 6456415. free.