Hexamethonium Explained

Hexamethonium is a non-depolarising ganglionic blocker, a neuronal nicotinic (nAChR) receptor antagonist[1] that acts in autonomic ganglia by binding mostly in or on the nAChR receptor, and not the acetylcholine binding site itself. It does not have any effect on the muscarinic acetylcholine receptors (mAChR) located on target organs of the parasympathetic nervous system, nor on the nicotinic receptors at the skeletal neuromuscular junction, but acts as antagonist at the nicotinic acetylcholine receptors located in sympathetic and parasympathetic ganglia (nAChR).[2]

Pharmacology

By blocking the neuronal nicotinic receptors in autonomic ganglia, which are necessary for transmission in all autonomic ganglia, both the sympathetic and parasympathetic nervous systems are inhibited. Its action on the neuronal nicotinic receptors is primarily through the block of the ion pore, rather than through competition with the binding site for acetylcholine.[3]

Postganglionic sympathetic systems are usually regulated by norepinephrine (noradrenaline) (adrenergic receptors), whereas parasympathetic systems are acetylcholine-based, and instead rely on muscarinic receptors (some post-ganglionic sympathetic neurons, such as those stimulating sweat glands, release acetylcholine).

The organ system and adverse effects of ganglion blockers are due to the parasympathetic and sympathetic stimuli blockage at preganglionic sites. Side-effects include combined sympatholytic (e.g., orthostatic hypotension and sexual dysfunction) and parasympatholytic (e.g., constipation, urinary retention, glaucoma, blurry vision, decreased lacrimal gland secretion, dry mouth (xerostomia)) effects.

Uses

It was formerly used to treat disorders, such as chronic hypertension, of the peripheral nervous system, which is innervated only by the sympathetic nervous system. The non-specificity of this treatment led to discontinuing its use.[4]

The use of inhaled hexamethonium, an unapproved drug, in a normal volunteer during a medical study is believed to have caused or contributed to her death[5] [6] in light of the presence of abnormal "ground glass opacities" on her chest X-ray.

See also

Notes and References

  1. Web site: Hexamethonium - Compound Summary. 2013-06-18 . PubChem . U.S. National Library of Medicine .
  2. Book: Howland RD, Mycek MJ . 2006 . Lippincott's illustrated reviews: Pharmacology . 3rd . 47 .
  3. Gurney AM, Rang HP . The channel-blocking action of methonium compounds on rat submandibular ganglion cells . British Journal of Pharmacology . 82 . 3 . 623–642 . July 1984 . 6146366 . 10.1111/j.1476-5381.1984.tb10801.x . 1987010 .
  4. Book: Hardman JB, Limbird LE, Gilman AG . Goodman and Gilman's The Pharmacological Basis of Therapeutics . 10th . 2001 . 210–211 . 978-0071354691 .
  5. Web site: Perkins E . Johns Hopkins' Tragedy: Could Librarians Have Prevented a Death? . August 7, 2001 . Information Today, Inc. . 2008-10-06.
  6. Savulescu J, Spriggs M . The hexamethonium asthma study and the death of a normal volunteer in research . Journal of Medical Ethics . 28 . 1 . 3–4 . February 2002 . 11834748 . 1733509 . 10.1136/jme.28.1.3 .