Indiplon Explained

Indiplon (INN and USAN) is a nonbenzodiazepine, hypnotic sedative that was developed in two formulations—an immediate-release formulation for sleep onset, and a modified-release (also called controlled-release or extended-release) version for sleep maintenance.

Pharmacology

Pharmacodynamics

Indiplon works by enhancing the action of the inhibitory neurotransmitter GABA, like most other nonbenzodiazepine sedatives. It primarily binds to the α1 subunits of the GABAA receptors in the brain.[1]

Pharmacokinetics

Indiplon has a short elimination half-life of 1.5 to 1.8 hours in young and elderly subjects, respectively.[2]

History

Indiplon was discovered at Lederle Laboratories (which was later acquired by Wyeth) in the 1980s and was called CL 285,489.[3] In 1998 Lederle licensed it, along with other early stage drug candidates, to DOV Pharmaceutical, a startup formed by former Lederle employees, and Dov exclusively sublicensed its rights in the drug to Neurocrine Biosciences in that same year.[3] In 2002, Neurocrine entered into an agreement with Pfizer to develop the drug.[3]

Indiplon was originally scheduled for release in 2007, when Sanofi-Aventis' popular hypnotic zolpidem lost its patent rights in the United States and thus became available as a much less expensive generic. In 2002, Neurocrine Biosciences had entered into an agreement with Pfizer to co-market indiplon in the US, in a deal worth a potential $400mn.[4] However, following the issuing of a non-approvable letter for the modified-release 15 mg formulation and an approvable letter with stipulations for the 5 mg and 10 mg immediate-release version by the FDA in May 2006,[5] Pfizer ended its relationship with Neurocrine.[6] Neurocrine's stock price dropped 60% on the news.[7]

Following a resubmission, the FDA in December 2007 deemed Neurocrine's new drug application (NDA) 'approvable' in the 5 and 10 mg formulations,[8] but requested new studies as a prerequisite to approval, including a clinical trial in the elderly, a safety study comparing adverse effects to those of similarly marketed drugs, and a preclinical study examining indiplon's safety in the third trimester of pregnancy.[9]

Following the 2007 FDA letter, Neurocrine decided to discontinue all clinical and marketing development of Indiplon in the United States.

External links

Notes and References

  1. Petroski RE, Pomeroy JE, Das R, Bowman H, Yang W, Chen AP, Foster AC . Indiplon is a high-affinity positive allosteric modulator with selectivity for alpha1 subunit-containing GABAA receptors . The Journal of Pharmacology and Experimental Therapeutics . 317 . 1 . 369–77 . April 2006 . 16399882 . 10.1124/jpet.105.096701 . 46510829 . PDF .
  2. Lemon MD, Strain JD, Hegg AM, Farver DK . Indiplon in the management of insomnia . Drug Design, Development and Therapy . 3 . 131–142 . September 2009 . 19920929 . 2769245 . 10.2147/dddt.s3207 . free .
  3. Book: Neubauer DN . Indiplon . 453–464 . GABA and Sleep: Molecular, Functional and Clinical Aspects. . Monti JS, Pandi-Perumal SR, Möhler H . Springer Science & Business Media . 2010 . 9783034602266 .
  4. Web site: San Diego's Neurocrine Biosciences Scores Second Big Deal in Two Days . . 18 June 2010 .
  5. Web site: Neurocrine's FDA Nightmare . TheStreet.com . 16 May 2006 .
  6. Web site: Pfizer Drops Neurocrine Deal . TheStreet.com . 22 June 2006 .
  7. Web site: Neurocrine stock price plunges 60 percent:FDA's mixed review of sleeping pill Indiplon could threaten Pfizer-Neurocrine partnership . CNN Money . 15 May 2006 .
  8. Neurocrine Receives Approvable Letter for Indiplon Capsules with Additional Safety and Efficacy Data Required by FDA. Neurocrine Biosciences, Inc.. 2007-12-13. 2007-12-13.
  9. Web site: Additional Pipeline Projects . Neurocrine . 2012-02-16 . https://web.archive.org/web/20120327204702/http://www.neurocrine.com/index.cfm?navId=25 . 2012-03-27 . live . 2014-06-24 .