Hemicholinium-3 Explained

Hemicholinium-3 (HC3), also known as hemicholine, is a drug which blocks the reuptake of choline by the high-affinity choline transporter (ChT; encoded in humans by the gene SLC5A7) at the presynapse. The reuptake of choline is the rate-limiting step in the synthesis of acetylcholine; hence, hemicholinium-3 decreases the synthesis of acetylcholine. It is therefore classified as an indirect acetylcholine antagonist.[1]

Acetylcholine is synthesized from choline and a donated acetyl group from acetyl-CoA, by the action of choline acetyltransferase (ChAT). Thus, decreasing the amount of choline available to a neuron will decrease the amount of acetylcholine produced. Neurons affected by hemicholinium-3 must rely on the transport of choline from the soma (cell body), rather than relying on reuptake of choline from the synaptic cleft.

Toxicity

Hemicholinium-3 is highly toxic because it interferes with cholinergic neurotransmission. The LD50 of hemicholinium-3 for mice is about 35 μg.[2]

See also

Notes and References

  1. Book: Carlson, Neil R. . vanc . 2007 . Physiology of Behavior . 9th . Boston . Pearson Education, Inc. . 978-0-205-46724-2 . 117.
  2. Freeman JJ, Kosh JW, Parrish JS . Peripheral toxicity of hemicholinium-3 in mice . British Journal of Pharmacology . 77 . 2 . 239–44 . October 1982 . 7139185 . 2044599 . 10.1111/j.1476-5381.1982.tb09291.x .