DNA-SCARS explained

DNA-SCARS (short for DNA segments with chromatin alterations reinforcing senescence) are nuclear substructures with persistent DNA damage and DNA damage response proteins found in senescent cells. DNA-SCARS are associated with PML nuclear bodies and the accumulation of activated ATM, ATR, CHK2 and p53 proteins. DNA-SCARS lack most of the characteristics of transient, reversible DNA damage foci, such as single-stranded DNA, active DNA synthesis, and DNA repair proteins RPA and RAD51.[1] Telomere dysfunction-induced foci (TIF) are generally associated with DNA-SCARS.

Together with senescence-associated heterochromatin foci (SAHF), DNA-SCARS are one of the most prevalent nuclear markers of cellular senescence.[2]

History

DNA-SCARS were discovered by Judith Campisi and colleagues, who first described them in 2011, although most of their characteristics were previously known.

Notes and References

  1. Rodier. F. Muñoz. DP. Teachenor. R. Chu. V. Le. O. Bhaumik. D. Coppé. JP. Campeau. E. Beauséjour. CM. Kim. SH. Davalos. AR. Campisi. J. DNA-SCARS: distinct nuclear structures that sustain damage-induced senescence growth arrest and inflammatory cytokine secretion.. Journal of Cell Science. 1 January 2011. 124. Pt 1. 68–81. 21118958. 10.1242/jcs.071340. 3001408.
  2. Rodier. F.. Campisi. J.. Four faces of cellular senescence. The Journal of Cell Biology. 14 February 2011. 192. 4. 547–556. 10.1083/jcb.201009094. 3044123. 21321098.